Well said, but I would add that there are many of us here (straight in my case) that have already had children (5 so far). I think I have done my job to populate the planet. If I chose to castrate myself (and my wife agrees) then it is my prerogative. I don't believe it will make me less of a man, it will make me the man that I want to be.Originally Posted by nonuts
There are no experts? But, yet you make these kinds of frightening, accusative, and hysterical statements?
The main problem I see with this thread is there is conversation about all sorts of things, there is no focus on one thing. The discussion here was ethyl alcohol, and yet experiences with lactic acid, and home made "chem cast" have been added to the mix as though they are comparative or even relevant. They are not.
Lactic acid is an acid, as acids do, they don't stop their action until they have become inert by dilution. Dilution is a result of it eating its way through whatever is there, until it reaches saturation. If one were to assume this would stop at say the wall of the testis, and not continue to eat it's way though until it was fully depleted, then one would be an idiot. So, point number one: Lactic Acid is NOT Alcohol, they do not have any common characteristics other than both being liquids.
Lactic acid remains active until it is diluted to inert levels.
Alcohol is metabolized by the body, assuming one's liver function is good enough to allow them to have an alcoholic beverage.
Chem-Cast (not to be confused with chemical castration drugs for temporary castration) was a chemical that was used for a short time in livestock (I believe it's out of production, since the cost/benefit wasn't competitive with other available methods). Even if the the discussion were about the chemical that was manufactured and sold for vet use, it was never approved for human use, never tested or researched for humans (there's really not a huge market for human castration, surprisingly enough). That this guy mixed up his own, based on some recipe he assumed he had correct, outside of a sterile process, and without ensuring total sterility, was begging for an infection. Injecting ANYTHING without using basic sterile precautions is asking for trouble.
Using a chemical that was never intended for human use, that was not made using sterile processes, with an unknown chemical equation (recipe) will very likely cause a serious infection.
Alcohol, especially pure medical grade alcohol (yes it exists, and is used in humans for the treatment of tumors and other procedures). Is sterile in its basic form, still the risk of infection exists, due to the process of injection, which is why anyone not knowing exactly what they are doing, should NEVER attempt ANY injection.
Finally, hyperplasia no one knows how cells divide, and reproduce as a result of inflammation or injury. There are many theories, but no absolutes. That it would be a causative for cancer is a real theoretical stretch. That alcohol is used to kill liver tumors suggests, this risk isn't that great. So, I don't think this does anything but add fear to the discussion.
Now, FEAR is a very healthy thing to have in this discussion, but let's try to keep the fear's relevant to the topic.
Number One: Never inject anything into your body, unless you are 100% confident you know what you are doing, 100% confident you are using a sterile product, and you know 100% what the risks and dangers are, and know what symptoms to be on the look out for that would indicate medical care.
Number Two: To be completely safe, just DON'T do it!
Number Three: Sadly the medical community doesn't support the desires of many here, they do desperate things, some of them foolish. If you decide to ignore number 2, please, please always have someone around to help, even if it's just to get you medical care.
OK, let's not let the bad experiences of some or the over-the-top replies of others let us lose focus on what this thread is about. I definitely wish to thank the management for being open-minded about this topic. It is definitely an emotional one for those involved, and the goal here is to share information so people DON'T do something terribly stupid and end up in the ER or worse. There are no clean 'n' easy surgical options on the continent where I live, and going to a cutter or doing it myself would be FAR worse.
There does need to be a conversation with respect to dosage and technique since these things are so important and we have a lot to learn from each other. 10 ml is a large dose which I did once; more typical is something in the 4 or 5 ml range. To mitigate against the stuff migrating around too fast, I was banded, and we kept an eye on the color and feeling of the surrounding tissues and skin. It is also very important to use a small needle, draw back first to make sure that no blood supply is involved, and NOT to force it at all. Also immediately stop at any sign of something strange; the membrane of the nut should be very good at holding in the alcohol and it is very elastic. In my case we'd anesthetize the cords first, so any sharp jolt of pain was a bad sign. We actually stopped one injection in progress on this basis; something was just not right.
After my first and second doses which were one day apart, they swelled up to be VERY large and the pain three days later was debilitating. Good thing I live in Germany where showing up to work is optional. They took a month and a half to go down to something slightly smaller than original, with a smaller round of injections in between. That got me down to 191 T, which my particular urologist did not think was low for a 30 year old man. Another round or two and a month and a half later, I came in at 312 T. Aaaaaaaaaagh. Other rounds of injections became far less debilitating but they're still very unpleasant.
With regard to the long-term risks of this sort of thing, the goal is to convince a doctor to remove them. I'm on testosterone therapy now, after a bit of cheating to get the T test below the magic number of 160 (my adrenals seem to be very active). So now they have NO use at all. There does seem to be a difference between countries in what a doctor would be willing to do; American doctors would at least think of removing a painful infarcted testicle while a German would tell you to go away unless it's obviously cancerous. If things get particularly bad I'll have to go to Thailand and fake some gender issues or, God forbid, get some help from a friend. I hope it doesn't come to this.
In conclusion, here's a list of what I think is important if you're going to attempt this.
1. Sterile technique! Infections suck.
2. Using high-quality ethyl alcohol, nothing of lower quality than everclear. Medical-grade is the best as it's used for killing tumors and has very few impurities.
3. Use a small needle; make sure you didn't hit the blood supply; and go very slowly. Banding them is not a bad idea either.
4. Anesthetizing the cord will make it easier to figure out if anything is happening outside of the nut, and definitely take it slow and listen to your body in any case.
5. Dosage-wise, your mileage will vary. There's a tradeoff among effectiveness, safety, and medium-term pain. 10 ml in my case was quite a bit; half that would be less effective but also would allow me to ride my bike to work again two days later.
6. Make sure you have a good GP and urologist lined up. As a 30 year old guy in a strange country I did not have either lined up; my first urologist's experience in my life was...odd...to say the least. The bedside manner was very Prussian, especially with the prostate check. I had to switch GPs too because my other GP was just not qualified to talk about male health. The current one is pretty good.
7. Be patient. I've heard stories of guys who've injected themselves and gotten doctors to remove them a few weeks later. I'd be lucky if I can get them removed, ever. Removal is the ultimate goal in my case, and it's easy to let this drive me nuts. This might mean that I have to keep injecting them to keep them from trying to grow back as I develop a plan B, but this sounds horrible too.
8. You should feel a bit of fear about this. It's completely healthy and it's a sign that you care about your life.
Well, yes... There are no experts on castration of humans via alcohol or injection of any other chemicals into the testicles. And, as a medical doctor I think my statements are appropriate.
First, I was not insinuating that the alcohol could or would burn through the tissue as lactic acid (or other caustics) can. I was stating that fistulas can form. Alcohol is a sclerosing agent. What that means is that it will dehydrate the cells and cause them to be damaged beyond repair. When those cells die off they leave holes which are either filled in by new scar tissue or in some cases stay patent by puss and debris. Sometimes these holes become fistulas (open passages between two or more organs). This is caused sometimes from infections as well, which could also be caused by injections.
Second, The vas deferens lie in areas that are very close to important structures. If you get a fistula from the vas to an artery the result could be deadly.
Nobody knows how cells divide? Please tell my biology professors and my genetics teachers. They are sadly mistaken that they think that they know how cells divide. I am sure they will tell you a little different. Please do a little reading prior to making statements like the above, there are more than just theories, there are basic scientific truths.
It is well known in the medical society that repeated damage is a causative agent for cancer. If you don't believe it look it up. Sun damage is the number one cause of skin cancer. How about liver cancer caused by heavy drinking? Yes due to repeated insults from overloading the liver. So I hope you can see that my statements are from sound medical judgment.
Liver tissue and testicular tissue are two seriously different tissues. Liver tissue dies all the time and replaces itself, where testicular tissue is not designed to do so.
Therefore, please don't accuse me of making "frightening, accusative, and hysterical statements," just because I have given advice in reason to my level of education. I believe that 10ml is too much volume to safely inject into a testicle (at least for most people).
Caveat emptor: Again as always please don't copy things you have seen here or anywhere else unless you have done the research and have made every attempt at being safe.
Everything I have learned in my medical training goes against performing self castration by any means. Please do not use any post that I make as medical reasoning to do anything of the such. Please do not PM me for medical advise, as I cannot legally give advice on this subject as I am not a urologist.
Last edited by saywhat; 11-16-2009 at 01:14 AM.
I am sorry to the learned doctor here, yes clearly you should have much more experience and medical training than I do. My mistake also for not being clear we were talking about how damaged cells divide, since it would depend on the cells, and how they were damaged, there would be no absolutes?
You said attempt suicide. That was not advice, and it was deeply insensitive, and certainly accusative.
Repeated sun damage causing skin cancer is well known, however, it is not established that the cause is due to the repeated damage or the repeated and excessive exposure to UV, or both.
Since 80% of liver cancer is caused by cirrhosis, and 40% of the people who die of cirrhosis each year have a history of alcohol abuse, there is for sure a link, but it's far from being caused from heavy drinking since only 10-20% (American Liver Foundation, who can't seem to narrow it down much more than with a huge margin of error) of heavy drinkers will develop cirrhosis.
Last edited by nonuts; 11-16-2009 at 03:53 PM.
You are right, that was poor wording on my part. I was just shocked at the volume that was injected. I meant to be frank, but not offensive. I am sorry.
I think we all could use a hug right now
http://deadon.files.wordpress.com/2007/07/group-hug.jpg
Hi I'm new here and I've been reading this post with great interest. However I do have a question regarding the type of over the counter alcohol used for the injections. Everyone here seems to agree to using "white" alcohol but can you use dark or brown if you will? Is there a primary reason for white VS brown. I would prefer to use some Jack and make those puppies happy while there bieng destroyed.
Secondly what would be the average time frame for total death to occur using 10mg injections in each testicle?
I think this is one thing we can agree on.
Obviously, the clear spirits, plain uncolored and unflavored vodka or Everclear will not have any coloring mater in them. Is anything colored bad? I don't know but why take a chance? Same with flavorings. I only know what I used and it worked so that my doctor diagnosed my testes as atrophied and infarcted and then removed them. I used smaller injections mainly 1 1/2 ml in each but I did get up to 6 ml once or twice. However my goal was not to see how much I could inject at once but just a steady pace with the end result in mind. My approached worked for me.
I don't think anyone can give you accurate "average" times because strength and dosage is varied and everyone seems to be doing this on their own and not part of any organized experiment.
Hugs are always GOOD!
It has to be DRINKING spirits only, color is not an issue, the higher the proof the better the results (less injections required for the result). Not anything but, DRINKING spirits other wise you can cause other damage to you self, up to and including death!
I had no problem with pain, but your milage may very.
_g
All said and done since mid may I have injected 68.5 cc's of everclear into each of my of my testicles. For the most par it was 1.5cc's at a time into both of them. I presently am waiting for the lupron to get out of my system so I can get blood work done to determine if I was sucessful or not. I will probally stop injecting around December to give myself time for maybe some shrinkage and also for the hematomas and some assorted bruising that has been occurring to heal. For me with the scaring from the clamping,all the injections and possibly some tissue damage due to the alcohol, bruising and hematomas occur more regularly. So far I am happy with the results. Both my testicles are very hard and misshapen. I am hopping for some shrinkage after I stop injecting for awhile. I keep planning to stop but it gets a little addicting after awhile and I want to be sure they are dead/
Maybe this is a good idea and maybe it's not. I just don't know. I'm not convinced that anyone hear understands how alcohol is metabolized and I kind of think you would be better off just using acetic acid (end by-product of alcohol metabolism). Just the same here is an article on alcohol metabolism:
www.ceri.com/alcohol.htm
Living with Alcohol
by Steven Wm. Fowkes
Alcohol is everywhere within Western culture. Since the dawn of recorded history, the art of fermenting fruit and grain into wine and beer has been a much-prized skill. As technology advanced, distillation of alcoholic beverages into refined spirits became quite popular. The high alcohol content of refined spirits made them exceptionally stable for long-term storage and commerce.
Along with fermentation technology came drunkenness, hangovers and alcoholism. Drunkenness is caused by alcohol’s pharmacological effect on the human central nervous system. This effect causes incoordination, slowed reaction time, muscle relaxation, behavioral disinhibition and impaired judgment, all of which last for several hours after alcohol is consumed. There is no simple and effective way to prevent drunkenness other than to avoid alcohol consumption in the first place.
Hangovers are the result of alcohol’s toxicity. Hangover symptoms include headaches, dehydration, irritability, sleep disturbances, liver toxicity, nerve and tissue hypersensitivity, etc. These symptoms can be prevented or significantly reduced by simple interventions that will be discussed in this article. These interventions augment natural detoxification mechanisms that would otherwise be overwhelmed by the sheer volume of alcohol intake.
Alcoholism can result from any one or combination of addictive mechanisms created by alcohol’s powerful pharmacological effect on the body. Some of these mechanisms are psychological and some are physiological. This article will discuss primarily physiological mechanisms. Although the majority of people consuming alcoholic beverages do not have problems with regulation of their intake, a significant minority of users do. Some learn to control their intake through adaptive behaviors. Many learn to cope through a strategy of abstinence. And some become alcoholics.
Acetaldehyde Toxicity
Alcohol’s effects are not limited to those of alcohol alone. Alcohol is metabolized in a multi-step process into various metabolites which have unique biochemical effects of their own. The first step in this process is the conversion of alcohol to acetaldehyde [see Figure A]. Since acetaldehyde is approximately 30 times more toxic than alcohol, acetaldehyde is a major cause of alcohol-associated side effects. If acetaldehyde is not efficiently converted into acetic acid (the second step in the metabolism of alcohol), severe toxicity can result. This is a common problem among certain people of Asian extraction (notably Innuit and American Indians) who have a genetic weakness in the acetaldehyde dehydrogenase enzyme [see Figure A]. Even in people who do not have this genetic trait, acetaldehyde dehydrogenase is often unable to fully keep up with the production of acetaldehyde during alcohol intoxication.
Cross-Linking
One of the most significant mechanisms of alcohol toxicity is the powerful cross-linking activity of acetaldehyde. Cross-linking is a process by which “molecular bridges” are formed between “reactive sites” on different molecules. These cross-links “tie up” the affected molecules and interfere with their normal function. In some circumstances, molecular function can be completely blocked by cross-linking.
A good example of the effect of cross-linking in action is the “tanning” of leather. The tanning process involves applying large amounts of a cross-linking agent (like tannic acid) to animal hides to cross-link the flexible collagen and elastin proteins in the animal skin to produce tough, inflexible, abrasion-resistant leather. This same process happens — at a slower rate — in people. Cross linking is largely responsible for age-related changes in human skin that make it inflexible, wrinkled and dry. Some of the most leathery skin you will ever see is found on alcoholics who stay outdoors a lot of the time. The ultraviolet (UV) and near-ultraviolet components of sunlight activate the cross-linking process.
There are many substances that can act as cross-linking agents. Aldehydes are one class of cross-linker, of which acetaldehyde is a member. Acetaldehyde is used in making plastics, adhesives and fabrics. The closely related chemical formaldehyde is used in insulating foams, plywood, particle board and embalming fluid.
Stewed, Not Pickled
The primary detoxification mechanism for scavenging unmetabolized acetaldehyde is sulfur-containing antioxidants [see Figure A]. The two most important are cysteine, a conditionally essential amino acid, and glutathione, a cysteine-containing tripeptide (a three-amino-acid polymer) [see Figure B]. Cysteine and glutathione are active against acetaldehyde (and formaldehyde) because they contain a reduced (unoxidized) form of sulfur called a sulfhydryl group, which contains a sulfur atom bonded to a hydrogen atom (abreviated SH).
Sulfhydryl groups interact with aldehydes to render tham incapable of forming cross links. This “mops up” or scavenges any stray acetaldehyde that is not properly metabolized into acetate (acetic acid) [see Figure A]. Although this is a powerful aldehyde detoxification mechanism, it is easily overwhelmed by the relatively large amounts of alcohol that are typically consumed with alcoholic beverages as compared to the amounts of alcohol and acetaldehyde that are produced through normal metabolism. Fortunately, sulfhydryl antioxidants can easily be fortified through dietary supplementation.
In one experiment with rodents [Sprince et al., 1974], a LD-90 dose of acetaldehyde (the dose that would normally kill 90% of the animals) was completely blocked by pretreatment of the animals with cysteine and vitamins B-1 and C. In other words, none of the cysteine-treated animals succumbed to the lethal dose of acetaldehyde! N-Acetylcysteine (NAC) protected almost as well as cysteine.
In another rodent experiment [Busnel & Lehman, 1980], alcohol’s ability to inhibit swimming after the alcohol had been completely metabolized was blocked by vitamin C. What this and the previous study suggests is that the pharmacologic and toxic effect of alcohol are different. The pharmacological effect (i.e., intoxication or drunkenness) is not inhibited by vitamin C or cysteine, but the toxic effect (e.g., the hangover, nervous irritability, swimming difficulty) is inhibited. This suggests that, with alcohol, you can “have your cake and eat it too.”
Dosage Suggestions
Typical doses of cysteine that are sufficient to block a major portion of the toxic effect of alcohol/acetaldehyde are about 200 mg per ounce of alcohol consumed. However, the rapid assimilation and metabolism of alcohol requires both prior and concurrent dosing of cysteine to maintain protection. Furthermore, a multifold excess of vitamin C is required to keep the cysteine in it’s reduced state and “on the job” against acetaldehyde. I use capsules (because they dissolve fast) containing 200 mg cysteine plus 600 mg of vitamin C (with or without extra B-1). I take one before I start drinking, one with each additional drink and one when I’m finished. It works remarkably well.
Additional Nutrients
There are several other nutrients which may synergize with cysteine and vitamin C. Glutathione, the predominant sulfhydryl antioxidant in the human body, should be considered. Although it is probably quite effective, it is many times more expensive than cysteine and it is not as concentrated; it contains only 10% sulfur compared to 26% sulfur in cysteine. Much larger doses of glutathione must be taken to get the same sulfhydryl concentration, and a significant but unknown amount of glutathione is broken down in the stomach into its component amino acids (glutamate, cysteine and glycine). So while glutathione is a great idea, it’s an expensive great idea.
Thiamine (vitamin B-1) and lipoic (thioctic) acid are key sulfur-containing nutrients that may be depleted by alcohol and/or may help with acetaldehyde detoxification. Thiamine was tested by Sprince and colleagues [1974] and found to offer protective benefit to acetaldehyde toxicity when combined with C and cysteine. Whether this is due to a direct interaction between acetaldehyde and the thiamine-bound sulfur or an enhancement of cellular energy production by the active thiamine cofactor (thiamine pyrophosphate) is not known. Alcoholics are known to be thiamine depleted, but whether this depletion is caused by diminished intestinal absorption of thiamine by alcohol or by destruction of thiamine by acetaldehyde is not known. Even under normal circumstances, intestinal absorption of thiamine is not very efficient.
In its reduced form, lipoic acid is a powerful sulfhydryl antioxidant. Due to lipoic acid’s twin sulfhydryl groups, it should scavenge aldehydes even more effectively that either cysteine or glutathione (see Figure B). However, supplemental lipoic acid is commercially available only in its oxidized form which contains no sulfhydryl sulfur. It is converted into the reduced form within the mitochondria after absorption from the bloodstream into the cell. So while lipoic acid may be a good cellular protector, it is not as efficient at scavenging acetaldehyde from the bloodstream as cysteine and glutathione. Lipoic acid is also fairly expensive.
Within the cells of the liver, however, lipoic acid and acetaldehyde may be readily interacting. The liver metabolizes the largest percentage of ingested alcohol and acetaldehyde levels may be quite high in liver cells. Acetaldehyde may bind to reduced lipoamide (the active lipoic acid factor) to render it inactive (see Figure B). Due to this potential problem, it may be a good idea not to take one’s regular dose of lipoic acid near when one drinks alcohol but rather several hours before and after.
Two combination formulas are available: Twin Labs and Vitamin Research Products make a C-and-cysteine formula, and VRP makes a B-1, C and N-acetylcysteine formula.
Addiction Mechanisms
The toxicity of acetaldehyde is mitigated to a significant extent by alcohol itself. This provides a strong incentive for people who start drinking alcohol to keep drinking alcohol. When they stop drinking, the toxic effects of acetaldehyde increase as the alcohol is rapidly cleared from the body. This mechanism reinforces “binge” drinking.
Blood Sugar Regulation
Alcohol also causes the liver to convert glycogen (a sugar-storage carbohydrate) into sugar. For people with blood-sugar regulation problems (primarily reactive hypoglycemia), alcohol can offer a “quick fix” to normalize blood sugar. This is why brandy is given to revive people who have fainted. The brain relies upon blood sugar for its primary energy supply.
For people with low blood sugar, alcohol can temporarily relieve their physiological and psychological symptoms. This provides a quick reward for alcohol consumption. If the effect of the alcohol is allowed to wear off, the symptoms return even stronger, providing additional incentive for further drinking. When alcohol consumption eventually stops, blood sugar tends to crash, severely agravating symptoms and distress. This might be described as “drinking one’s self into unconsciousness.” This problem leads to binge drinking behaviors.
The high prevalence of blood sugar-related alcohol addictive behaviors is supported by the high percentage of former alcoholics who have become “sugar junkies.” It is probably no accident that Alcoholics Anonymous meetings and 12-step programs usually feature ample availability of sweet snack foods (cookies, cakes, candy, soft drinks, etc.).
The solution to this problem is improved glycemic control. This can be accomplished by such dietary changes as eating complex carbohydrates instead of simple carbohydrates, eating less carbohydrates (carbohydrate restriction) to activate fat-burning enzymes, and eating smaller, more frequent, high-protein meals. It can also be addressed with supplements. The amino acid glutamine, for example, provides an alternative source of fuel for the brain. Supplemental glutamine can make the brain less sensitive to low blood sugar. Typical doses are 1-3 g.
The trace mineral chromium is an essential part of glucose tolerance factor (GTF), which is necessary for efficient transport of blood sugar into the insulin-sensitive cells of the body. Chromium is slow to absorb and accumulate, so extended use is required. Chromium chloride, chromium nicotinate and chromium picolinate are commonly available as supplements. Typical high-potency chromium supplements are in the 200 mcg range, although many physicians will recommend that their diabetic and diabetic-prone patients take 1000 mcg. Typical maintenance doses are in the 25-50-100 mcg range.
The NADH Connection
When alcohol is metabolized, it has a powerful effect on cellular energy production pathways. The conversion of alcohol into acetaldehyde by alcohol dehydrogenase and acetaldehyde into acetate by acetaldehyde dehydrogenase results in the generation of NADH (reduced nicotinamide adenine dinucleotide). NADH is the hydrogen-transfer chemical (i.e., electron transporter) that enables oxidative phosphorylation to take place (i.e., the production of ATP energy within the mitochondria through the utilization of oxygen). This process is too complex to discuss in detail here, but an etensive review article can be found in Smart Drug News Volume 5, No. 2.
Suffice it to say that NADH production generates lots of energy. This is why drinking alcohol warms people up. The extremely rapid NADH production from the alcohol dramatically increases energy availability and body temperature, especially in people who are chilled. After alcohol has been converted into acetate, the acetate enters the citric acid cycle to generate even more NADH. Vinegar is about 5% acetic acid (acetate).
For people with compromised mitochondrial function, alcohol may provide a temporary shot of energy that can energize the brain for dealing with stressful circumstances. This increased energy, combined with muscle relaxation and behavioral disinhibition, can be percieved as a valuable aid to social interaction.
Mitochondrial energy production can be supported through diet and supplements instead of alcohol. NADH is now available as a supplement in 2.5 and 5 mg doses. Dosages range from 2.5 mg to more than 10 mg. NAD (the oxidized form of NADH) is also available as a supplement. NAD is made in the human body in two ways: primarily from the essential amino acid L-tryptophan, and secondarily from niacinamide (vitamin B-3). Both of these can be taken supplementally.
In addition to making NAD, tryptophan also makes serotonin, a brain neurotransmitter involved in the regulation of carbohydrate feeding behaviors, mood and emotional control. Elevated serotonin levels suppress the appetite for carbohydrates (“sugar cravings”) and promote calmness and equanimity. Low serotonin levels increase carbohydrate appetite, aggressive and violent behaviors, and depression. Maybe this factor is involved in the difference between “happy” and “angry” drunks.
The natural production of NADH (in the absence of alcohol) depends on lipoic acid, thiamine and riboflavin. Since lipoic acid and thiamine contain sulfur which may become bound to acetaldehyde, I wonder whether the use of alcohol compromises the very energy pathways it stimulates.
That’s it for the lesson. Now it’s time to practice. Have fun! But, please, don’t forget the designated driver.
][- Ç ]> Heta, Stigma, Sho. The lost, rejected, outcaste and forgotten Greek Letters Society.
(Sorry no numeric keypad)
Here is my update.
Over the last few weeks, I ultimately injected each testicle 10 times with 1 cc per teste each time with about 2 days inbetween injections.
I have not injected for a week now, and I have no nasty side effects at this point in time. The one effect that I note is a dramatic decrease in labido/sex drive. That effect is the one that I was striving for and so I think that the program worked and was effective. I believe that the process is complete for me and that it only took 10 (or less - - - who knows) injections to do the job.
I will be able to prove this one way or the other sometime around Jan of 2010 when I have my next endochronologist appointment. Since I am on Premarin, he always tests my blood for T and E and I have a 10 year history of baselines for these hormone levels. That test will be the real proof of the pudding (no pun intended).
Danielle
Last edited by Danielle; 11-19-2009 at 07:54 PM.
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